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Answer :
In individuals with normally functioning androgen receptors and a 46-XY karyotype, the presence of functional androgen receptors allows for the feedback regulation of luteinizing hormone (LH) production.
LH is involved in stimulating the production of testosterone in the testes. However, in a person with a mutation in their androgen receptor gene that renders their receptors non-functional, the feedback loop is disrupted. Without functional androgen receptors, the body is unable to sense the presence of testosterone. As a result, the hypothalamus and pituitary gland may perceive low testosterone levels and respond by increasing LH production.
Therefore, individuals with non-functional androgen receptors may exhibit elevated LH levels compared to those with normally functioning receptors.
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Complete Question:
A person who is 46-XY has a mutation in their androgen receptor gene that renders their androgen receptors completely non-functional. How would their luteinizing hormone (LH) levels appear relative to individuals with normally functioning androgen receptors who also have a 46-XY karyotype? Please explain the reason behind this difference.
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