We appreciate your visit to In crosses between fish of two different morphologies the lack of bony armor segregates as a recessive trait that maps to the ectodermal dysplasin eda. This page offers clear insights and highlights the essential aspects of the topic. Our goal is to provide a helpful and engaging learning experience. Explore the content and find the answers you need!
Answer :
Epigenetic variation is probably present as a result of exposure to predators. The CpG island ahead of a Eda gene inside the non-spiny fish is probably methylated in the DNA.
Although epigenetic modifications can affect how your body interprets a DNA sequence, they are reversible and don't alter your DNA sequence.
Epigenetics is the investigation of how environmental factors and behavior may alter how your genes function. While epigenetic alterations are reversible and don't alter your DNA sequence like genetic changes do, they can alter how your body interprets a DNA sequence.
Epigenetic alterations to DNA control whether or not genes are activated. These alterations are connected to DNA and don't alter the order in which DNA is built.
Learn more about epigenetic variation at
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The question is -
Three-spined stickleback fish live in lakes formed when the last ice age ended 10,000 to 15,000 years ago. In lakes where the sticklebacks are prey for larger fish, they develop 35 bony plates along their body as armor. In contrast, sticklebacks in lakes where there are no predators develop only a few or no bony plates. In crosses between fish of the two different morphologies, the lack of bony armor segregates as a recessive trait that maps to the ectodermal dysplasia (Eda) gene. Comparisons between the Eda-coding regions of the armored and non-armored fish revealed no differences. How can you explain this result?
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