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Answer :
After inhalation of carbon monoxide (CO), the following observations would be expected:
Decreased alveolar PO2: Carbon monoxide has a higher affinity for hemoglobin than oxygen, leading to the formation of carboxyhemoglobin.
This reduces the amount of oxygen that can bind to hemoglobin, resulting in decreased alveolar partial pressure of oxygen (PO2).
Increased alveolar PCO2: Carbon monoxide does not directly affect the partial pressure of carbon dioxide (PCO2). However, CO can impair oxygen transport and lead to tissue hypoxia. In response to hypoxia, the body increases ventilation to compensate, which results in increased alveolar PCO2.
Greater activity in peripheral chemoreceptors: Peripheral chemoreceptors, located in the carotid and aortic bodies, sense changes in arterial blood oxygen levels. When exposed to carbon monoxide, which reduces oxygen transport, the peripheral chemoreceptors become more active in an attempt to restore oxygen levels.
Decreased hemoglobin concentration of blood: Carbon monoxide binds strongly to hemoglobin, forming carboxyhemoglobin. This reduces the amount of functional hemoglobin available for oxygen transport, leading to decreased hemoglobin concentration in the blood.
It is worth noting that carbon monoxide poisoning can have various symptoms depending on the severity and duration of exposure. These symptoms may include headache, dizziness, confusion, nausea, shortness of breath, and even loss of consciousness or death in severe cases. If you suspect carbon monoxide poisoning, it is important to seek immediate medical attention.
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